Association of Amyloid Pathology With Myelin Alteration in Preclinical Alzheimer Disease.

Aging and Cognitive FunctionsAlzheimer’s DiseaseMultiple Sclerosis

Alzheimer disease pathology and the cerebrospinal fluid proteome

Alzheimers Res Ther.

Dayon L, Núñez Galindo A, Wojcik J, Cominetti O, Corthésy J, Oikonomidi A, Henry H, Kussmann M, Migliavacca E, Severin I, Bowman GL, Popp J

doi: 10.1186/s13195-018-0397-4.

Alzheimer’s Disease

Association of Cortical β-Amyloid Protein in the Absence of Insoluble Deposits With Alzheimer Disease

JAMA Neurol.

Yu L, Petyuk VA, Tasaki S, Boyle PA, Gaiteri C, Schneider JA, De Jager PL, Bennett DA

doi: 10.1001/jamaneurol.

COMMENTED

Alzheimer’s Disease

Poor sleep is associated with CSF biomarkers of amyloid pathology in cognitively normal adults

Neurology.

Sprecher KE, Koscik RL, Carlsson CM, Zetterberg H, Blennow K, Okonkwo OC, Sager MA, Asthana S, Johnson SC, Benca RM, Bendlin BB

doi: 10.1212/

Aging and Cognitive FunctionsAlzheimer’s Disease

Circadian Rest-Activity Pattern Changes in Aging and Preclinical Alzheimer Disease

JAMA Neurol.

Musiek ES, Bhimasani M, Zangrilli MA, Morris JC, Holtzman DM, Ju YS

doi: 10.1001/

Aging and Cognitive FunctionsAlzheimer’s DiseaseDementia

Association Between Amyloid and Tau Accumulation in Young Adults With Autosomal Dominant Alzheimer Disease

JAMA Neurol.

Quiroz YT, Sperling RA, Norton DJ, Baena A, Arboleda-Velasquez JF, Cosio D, Schultz A, Lapoint M, Guzman-Velez E, Miller JB, Kim LA, Chen K, Tariot PN, Lopera F, Reiman EM, Johnson KA

doi: 10.1001/jamaneurol.

COMMENTED

Aging and Cognitive FunctionsAlzheimer’s Disease

Prevalence of preclinical Alzheimer disease: Comparison of current classification systems

Neurology.

Kern S, Zetterberg H, Kern J, Zettergren A, Waern M, Höglund K, Andreasson U, Wetterberg H, Börjesson-Hanson A, Blennow K, Skoog I

doi: 10.1212/

Aging and Cognitive FunctionsAlzheimer’s DiseaseDementia

Linking Amyloid-β and Tau Deposition in Alzheimer Disease

JAMA Neurol.

Vemuri P, Schöll M

doi: 10.1001/

Aging and Cognitive Functions

Distinct spatiotemporal accumulation of N-truncated and full-length amyloid-β42 in Alzheimer's disease

Brain.

Shinohara M, Koga S, Konno T, Nix J, Shinohara M, Aoki N, Das P, Parisi JE, Petersen RC, Rosenberry TL, Dickson DW, Bu G

doi: 10.1093/brain/awx284.

Aging and Cognitive FunctionsDementia

Prominent Non-Memory Deficits in Alzheimer's Disease Are Associated with Faster Disease Progression

J Alzheimers Dis.

Scheltens NME, Tijms BM, Heymans MW, Rabinovici GD, Cohn-Sheehy BI, Miller BL, Kramer JH, Wolfsgruber S, Wagner M, Kornhuber J, Peters O, Scheltens P, van der Flier WM, Amsterdam Dementia Cohort, Alzheimer’s Disease Neuroimaging Initiative, German Dementia Competence Network, University of San Francisco Memory and Aging Center

doi: 10.3233/JAD-171088.

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